<human>: Condense the text into a few key points

my grandmother had just died three days ago. She was 78, had diabetes, and dementia.  
Background:
Last month she was admitted to the hospital with protein-deficiency, and thus edema, and generally just weak. She was treated with albumin, and then human serum albumin, and antibiotics such as meroxan, meroven.  
During her last days at the ICU, she had liver and kidney failure, and mucus/phlegm drowning her lung, she stopped breathing so many times. (On the last day she stopped breathing 30+ times, kept being revived by ICU staff with chest pumps and adrenaline as her heart was still pumping and pupils were still responding to light).  
Due to the protein-deficiency, some in our family suspect her caretaker did not feed her enough protein (from what I observed her daily diet, made by the caretaker, consisted of carbs, with some vegetable soup).  
But during the wake, a family friend who is a doctor raised a possibility that she might have been frequently given CTM (Chlor-trimeton, trade name for chlorphenamine), to sedate her. Now this antihistamine drug has been known to be the drug of choice for nannies in my country to sedate babies and kids, to make their job easier. There have been many criminal cases involving CTM filed around these parts.  
My question is,
<bot>: could long-term use of chlorphenamine have caused directly/indirectly her albumin production and her liver in general to deteriorate?